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Cigarette smoking is the most important preventable cause of
premature death in the United States. It accounts for nearly 440,000 of
the more than 2.4 million annual deaths. Cigarette smokers have a higher
risk of developing several chronic disorders.
These include fatty build ups in arteries, several types of cancer
and chronic obstructive pulmonary disease (lung problems).
Atherosclerosis (build up of fatty substances in the arteries) is a
chief contributor to the high number of deaths from smoking. Many
studies detail the evidence that cigarette smoking is a major cause of
coronary heart disease, which leads to heart attack.
Bronchitis and Emphysema
Chronic bronchitis and emphysema are two common lung diseases
specifically associated with smoking.
Chronic bronchitis is the production of excess mucus in our lungs and
causes problems in absorbing oxygen. The excess mucus impairs breathing
and is usually associated with constant coughing, which is a natural
mechanism that removes foreign particles and irritants from our lungs.
Healthy lungs have a natural cleaning process that removes the dust
and smoke and other foreign particles that are part of the air, allowing
us to breathe normally.
Tobacco smoke contains many harmful chemicals, and the smoke itself
limits our lungs’ natural filtering or cleaning system. Cigarette smoke
also contains substances that directly damage the lung tissue.
Smoking irritates the bronchial tubes in the lungs which in turn
results in the production of more mucus. Smokers’ cough, as it is
commonly known, is a sign that the lungs are trying to clean themselves.
Long-term exposure to cigarette smoke destroys the structure of our
lungs, reducing their capacity to absorb oxygen. At the same time, the
tiny airways which transfer oxygen from the lungs to the blood vessels
are destroyed, resulting in less oxygen getting into the bloodstream.
The symptoms of chronic bronchitis are a constant cough and excess
phlegm. There is also a higher incidence of throat and lung infections.
People with chronic bronchitis are more susceptible to both lung and
heart failure.
Unless treated, the disease will become progressively worse.
Quitting smoking will stop the build-up of excess mucus and phlegm.
After quitting, smokers will find that they cough more than when they
smoked, but this is due to the cleaning mechanism working to clear out
the accumulated smoke-related mucus and phlegm. This is only temporary.
Emphysema
Emphysema, often called “lung rot”, is a degenerative disease.
It shows up as shortness of breath - a feeling of breathlessness
accompanying any movement or exertion.
The first signs of emphysema may only be a slight shortness of
breath. Many people consider their heavy breathing as just being out of
shape, but gradually they will notice that climbing stairs or lifting
heavy objects makes them wheeze and leaves them breathless.
As the disease progresses, breathing becomes a major effort. Nothing
can be done to correct the damage caused by emphysema. Lung damage is
permanent and cannot be reversed.
Emphysema is a debilitating disease and as individuals age, they
become progressively weaker.
Nearly all cases of emphysema are caused by long-term smoking.
Those who smoke approximately 20 cigarettes a day will, sooner or
later, have some sign of the disease
Stopping smoking will help stop the progression of the disease.
What You Can Do
With both bronchitis and emphysema, the first step in preventing
further lung damage is to quit smoking.
Mouth diseases
Smoking tobacco, whether it’s cigarettes, cigars or pipes, increases
the risk of cancer on the lips, in the mouth or in the throat.
Depending on the number of cigarettes smoked, cigarette smokers are
six times more likely to die from different mouth related cancers.
Cigar smokers are twice as likely to die from mouth related deceases
compared to non-smokers. The most common places where oral cancer forms
is on the tongue, the gums, or the floor of the mouth.
Using chewing tobacco also increases the risks of mouth and throat
cancers
Impotence and smoking
Impotence, or erectile dysfunction, is twice as likely to occur in
smokers than non-smokers. Exposure to second-hand smoke is a significant
factor in becoming impotent.
Impotence is the constant inability of a man to maintain an erection
for sexual purposes. It is estimated that impotence affects over 30
million men in the United States.
Sexual functioning requires the coordination of several functions in
the body. This includes the nervous system (mental stimulation) working
in coordination with hormones, and the vascular system which pumps blood
into the muscle tissue that maintains the erection. Smoking can affect
all these systems, thereby, causing impotence.
Many diseases caused by smoking involve blockage of the arteries,
meaning that smoking inhibits the flow of blood throughout the body.
Strokes and heart attacks are one result of severe blood flow blockage.
Impotence is another result of arterial blockage.
Long-term smoking causes more physiological damage. In a United
States study, a healthy men who smoked, with no history of impotence,
heart disease or diabetes, were interviewed twice over 8 years. The
second interview revealed that although these men still had no heart
disease or diabetes, they were twice as likely to experience moderate or
severe impotence.
A secondary study indicated that smoking is an independent risk
factor contributing to impotence. After considering other risk factors,
it was found that current smokers were twice as likely to experience
impotence as non-smokers.
The study concluded: The heavy smokers (40+ cigarettes/day) had the
“softest” night-time erections; smoking was related to an abnormal
decline of blood pressure in the penis.
It is possible to fully or partially recover erectile function by
just quitting smoking. The problem in recovery is the degree of damage
that has been inflicted, and the identification of which system has been
affected. The greater the length of time spent smoking, the greater the
damage to the biological systems that cause impotence. Men who have
reported losing their erections before orgasm have completely reversed
this situation by quitting smoking.
Smoking is a major risk factor that contributes to a form of heart
disease called cardiovascular disease. Cardiovascular disease is a major
cause of death in Canada.
Cardiovascular disease
Cardiovascular disease is defined as diseases and injuries of the
heart, the blood vessels of the heart, and the system of blood vessels
(veins and arteries) throughout the body and within the brain.
Cigarette and tobacco smoke, high blood cholesterol, high blood
pressure, physical inactivity, obesity and diabetes are the six major
independent risk factors for coronary heart disease that you can modify
or control. Cigarette smoking is so widespread and significant as a risk
factor that the Surgeon General has called it “the leading preventable
cause of disease and deaths in the United States.”
Cigarette smoking increases the risk of coronary heart disease by
itself. When it acts with other factors, it greatly increases risk.
Smoking increases blood pressure, decreases exercise tolerance and
increases the tendency for blood to clot. Smoking also increases the
risk of recurrent coronary heart disease after bypass surgery.
Cigarette smoking is the most important risk factor for young men and
women.
It produces a greater relative risk in persons under age 50 than in
those over 50. Women who smoke and use oral contraceptives greatly
increase their risk of coronary heart disease and stroke compared with
nonsmoking women who use oral contraceptives.
Smoking decreases HDL (good) cholesterol. Cigarette smoking combined
with a family history of heart disease also seems to greatly increase
the risk.
Although there are many aspects of this disease, the fundamental
problem is that the supply of oxygen and the necessary nutrients carried
by our blood are constricted or blocked. This causes injury to our heart
muscles. Smoking, or even exposure to second-hand smoke, makes the heart
work harder by:
Decreasing the oxygen carried in the blood - carbon monoxide and
other gases replace oxygen with each inhalation of cigarette smoke;
Increasing the heart rate - the heart beats faster to get more oxygen by
accessing a greater volume of oxygen-poor blood;
Decreasing the size of blood vessels - a build up of fat deposits
associated with nicotine and carbon monoxide makes blood vessels and
arteries smaller which limits the blood supply to the heart.
Cigarette smoking is a well-known risk factor that contributes to,
and significantly, increases the chance of having cardiovascular
disease. In 1996, cardiovascular disease accounted for 37% of all deaths
in Canada.
While more men than women died of heart disease (22% versus 19%),
more women died of stroke (9% versus 6%). The build up of fat deposits
in the blood supply system is called Coronary Artery Disease (sometimes
called Coronary Heart Disease or C.H.D.) and can eventually lead to
blocked blood vessels and arteries, which, in turn, can cause permanent
damage to the heart muscles.
The damage to the heart muscles because of a lack of oxygen can
result in a heart attack.
The risk of developing Coronary Heart Disease increases with the
length and intensity of exposure to cigarette smoke
Smokers have a 70% greater chance of dying from Coronary Heart
Disease than non-smokers.
Smoking more than 40 cigarettes per day increases the chance of dying
by 200 to 300% in comparison with non-smokers.
Among people younger than 65, 45% of Coronary Heart Disease in men
and 40% in women is caused by cigarette smoking.
Among people 65 or older, 15% of Coronary Heart Disease in men and 9%
in women is caused by smoking.
Stopping smoking reduces the risk of smoking-related cardiovascular
disease by approximately 50% within one year, and to normal levels (i.e.
people who never smoked) within five years.
Smoking and strokes
Cardiovascular disease is the major cause of death in Canada and
affects two organs in the body: the heart and the brain. When the brain
is affected, it is called a stroke, which is a blockage in the blood
vessels that limits or stops the blood flow to or in the brain.
Studies show that cigarette smoking is an important risk factor for
stroke.
Inhaling cigarette smoke produces several effects that damage the
cerebrovascular system. Women who take oral contraceptives and smoke
increase their risk of stroke many times. Smoking also creates a higher
risk for peripheral arterial disease and aortic aneurysm.
A stroke is a brain injury caused by a lack of blood.
Without blood, the brain does not receive the necessary nutrients and
oxygen. This can lead to permanent damage.
Having a stroke can lead to severe mental or physical problems, such
as full paralysis, partial paralysis, loss of speech or loss of sight.
There are three types of stroke: transient ischemic attack (TIA),
haemorrhagic, and ischemic:
A TIA is a mini-stroke. Generally, there is no permanent brain
damage, but it is often a warning that a major stroke is about to
happen.
Seeing a doctor immediately lessens the risk of a major stroke;
Haemorrhagic strokes occur when a blood vessel bursts inside the brain,
increasing pressure in the head and injuring brain cells;
An ischemic stroke is the most serious and permanently damages brain
cells by starving them of necessary oxygen and nutrients.
Cigarette smoking is a well-known risk factor that contributes to,
and significantly increases, the chances of having a stroke. The risk of
stroke is approximately 50% higher in smokers than in non-smokers. The
risk increases with the number of cigarettes smoked per day.
Smokers who consume more than 25 cigarettes/day have the highest risk
of a stroke. Stopping smoking reduces the risk of stroke:
by approximately 50% within one year;
to normal levels (people who never smoked) within five years.
Source : Internet |